You also suppose that myopia and/or astigmatism could born outside of the eye and/or brain - and that’s a brilliant idea I ended up my research with.
Don’t get me wrong - that does not mean the stimulus and reponse don’t work.
But another idea if axial elongation is from muscle action is that eye, just like every part of body can compensate and then it gets stuck in a pattern. Why? Because some muscles are shorter and some are longer due to deformed skull - and every time eye gets muscle work (accommodation), every time it reinforces that pattern. Bates’ exercises were all about getting out of the pattern. EndMyopia is about retraining the neuromuscular system to fire properly, the question is why the eye, while being incomparably lighter than a bone takes too long to change by a negligible amount. And seeking the faster improvement, even than 1.25 D/year is not any shortcut, no matter how much the myopia is. It’s enough for the tecnique to be cause-addressing, and I aimed with passion for EM to show non-folky science, in years of heavy enough funding, isn’t the time @jakey ? It was all about reverting the eye back to its premyopic shape, and the community would wonder even about the possibility of this - definitely the lack of materials proving that.
Digging into the relationship about EOMs and myopia, EOM paths and skull deformities is a great field in occupation in eye research. But I am happy with what I have (also things besides EM) and rather let someone other will do that job. Becuase the treatment would differ, depending on which kind of cause is behing axial elongation - neuromuscular/biomechanical or metabolical/biochemical.